Autism, What Can We Do? 04/03/2014 0 Comments   The prevalence of autism in the United States is on the rise according to numbers released by the Center for Disease Control and Prevention (CDC). The chart below shows a dramatic rise over the last 37 years of children diagnosed with Autism Spectrum Disorder, which is 2012 was believed to be 1 in 88 children. That is OLD data, the CDC recently published  Prevalence of Autism Spectrum Disorder Among Children Aged 8 Years published on March 28, 2014 About 1 in 68 children has been identified with autism spectrum disorder (ASD) according to estimates from CDC's Autism and Developmental Disabilities Monitoring (ADDM) Network.  ASD is almost 5 times more common among boys (1 in 42) than among girls (1 in 189) ASD tends to occur more often in people who have certain genetic or chromosomal conditions. About 10% of children with autism are also identified as having Down syndrome, fragile X syndrome, tuberous sclerosis, or other genetic and chromosomal disorders.[7-10] Almost half (46%) of children identified with ASD has average to above average intellectual ability. American Journal of Medical Genetics Part B: Neuropsychiatric Genetics published a study called: Metabolic endophenotype and related genotypes are associated with oxidative stress in children with autism†Autism is a behaviorally defined neurodevelopmental disorder usually diagnosed in early childhood that is characterized by impairment in reciprocal communication and speech, repetitive behaviors, and social withdrawal.(13) Although both genetic and environmental factors are thought to be involved, none have been reproducibly identified. The metabolic phenotype of an individual reflects the influence of endogenous and exogenous factors on genotype. As such, it provides a window through which the interactive impact of genes and environment may be viewed and relevant susceptibility factors identified.  They concluded that  an increased vulnerability to oxidative stress (endogenous or environmental) may contribute to the development and clinical manifestations of autism.(13) The International Journal of Neuropsychopharmacology published a study  Oxidative stress in psychiatric disorders: evidence base and therapeutic implications which they reported: Oxidative stress has been implicated in the pathogenesis of diverse disease states, and may be a common pathogenic mechanism underlying many major psychiatric disorders, as the brain has comparatively greater vulnerability to oxidative damage. This review aims to examine the current evidence for the role of oxidative stress in psychiatric disorders, and its academic and clinical implications. A literature search was conducted using the Medline, Pubmed, PsycINFO, CINAHL PLUS, BIOSIS Previews, and Cochrane databases, with a time-frame extending to September 2007. The broadest data for oxidative stress mechanisms have been derived from studies conducted in schizophrenia, where evidence is available from different areas of oxidative research, including oxidative marker assays, psychopharmacology studies, and clinical trials of antioxidants. For bipolar disorder and depression, a solid foundation for oxidative stress hypotheses has been provided by biochemical, genetic, pharmacological, preclinical therapeutic studies and one clinical trial. Oxidative pathophysiology in anxiety disorders is strongly supported by animal models, and also by human biochemical data. Pilot studies have suggested efficacy of N-acetylcysteine in cocaine dependence, while early evidence is accumulating for oxidative mechanisms in autism and attention deficit hyperactivity disorder. In conclusion, multi-dimensional data support the role of oxidative stress in diverse psychiatric disorders. These data not only suggest that oxidative mechanisms may form unifying common pathogenic pathways in psychiatric disorders, but also introduce new targets for the development of therapeutic interventions. (14) Oxidative stress is at the basis of over 200 diseases and counting. We have found one way that reduces oxidative stress  significantly, it natural, no side effects or drug interactions. It's called Protandim, which is a Nrf2 activator. Take it once a day and between day 30-60 many notice changes. Clinical studies should be out in the near future. Order it HERE  Thinking nutrition or changing the diet will help? You may see some changes but look at the chart above. The top three Antioxidants are not ones you can eat, your body makes them.  You will also notice the absorption rates vary by each person. The American Society for Clinical Nutrition published a study that describes Autism as a complex neurodevelopmental disorder that usually presents in early childhood and that is thought to be influenced by genetic and environmental factors. Although abnormal metabolism of methionine and homocysteine has been noted in autisim. (1) In the study, relative to the control children, the children with autism had significantly lower baseline plasma concentrations of methionine, SAM, homocysteine, cystathionine, cysteine, and total glutathione and significantly higher concentrations of SAH, adenosine, and oxidized glutathione. This metabolic profile is consistent with impaired capacity for methylation (significantly lower ratio of SAM to SAH) and increased oxidative stress (significantly lower redox ratio of reduced glutathione to oxidized glutathione) in children with autism. The intervention trial was effective in normalizing the metabolic imbalance in the autistic children.(1) Conclusions: An increased vulnerability to oxidative stress and a decreased capacity for methylation may contribute to the development and clinical manifestation of autism.(1) Never heard of oxidative stress? It's what biochemically ages us, its free radical damage to our cells gone mad. It'sOxidative stress is essentially an imbalance between the production of free radicals and the ability of the body to counteract or detoxify their harmful effects through neutralization by antioxidants. Food or antioxidant supplements dont come close to helping the body regulate free radicals and oxidative stress. Your OWN BODY can produce up to a million antioxidant enzymes (as seen on the top of the chart above) at 1 million, per second, every second.  The difference is staggering. Let your own body do what it does bes naturally. The way to activate your own bodies master switch (NRF2) is with a Nrf2 activator with proven peer reviewed science, like inProtandim. Types of Treatments  There are many different types of treatments available. For example, auditory training, discrete trial training, facilitated communication, music therapy, occupational therapy, physical therapy, and sensory integration. The different types of treatments can generally be broken down into the following categories: Behavior and Communication Approaches Dietary Approaches Medication Complementary and Alternative Medicine A  natural therapy is found in reducing oxidative stress. Oxidative stress is a slow death of cells which affect DNA and genes. Current studies are underway but are finding reduction of oxidative stress, and specifically through activating the Nrf2 pathway which turns on our antioxidant enzymes, can reduce the oxidation (cell death) by 40% on average and up to 70%. Some reports show that children are able to decrease other medications and have behavioral improvements.  Order  Protandim NOW and get a 20% discount.